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In nerves uncovered to 100 Hz when bathed in 10 mM glucose, the CAP failed suggesting that even in the presence of normoglycemic glucose extra vitality substrate is required when the tissue energy demand increases. Such knowledge are important as they present that glycogen has an essential role under physiological circumstances. The truth that glycogen supported CAP conduction in the absence of exogenously utilized glucose in all probability has no value in ascribing a task to glycogen, since aglycaemia is an synthetic experimental process, and hypoglycemia is an iatrogenic consequence of insulin therapy that did not exist previous to 1922 (Frier et al., 2014), thus can't be glycogen’s predominant role. However supplying lactate to axons when tissue energy demand increases implies a novel role for mind glycogen and suggests it acts as an power buffer somewhat than an power reserve, whose function is to not single-handedly support conduction, but to act in live performance with ambient levels of normoglycemic glucose.

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